This morning, Axeon boss Axel Merckx announced that Young was not expected to recover: “At this time, his family asks for privacy,” team manager Axel Merckx said. Why You Need To Stop Worrying About The Color Of Your Ketostix. Yeah, I know you like to use them, but there are so many misconceptions about what they are telling you, that I need to intervene and make sure you get it. But before I go there, let me urge you to just buy The Art and Science of Low Carbohydrate Living, and read pages 1. If I could copy these pages verbatim and paste it here, I would. Their caloric content is thereby wasted. Of the three types of ketones (acetate, acetoacetate, and beta- hydroxybutyrate) produced by your body, ketostix only measure acetoacetate. As mentioned before, beta- hydroxybutryrate is not technically a ketone. When plasma acetoacetate concentrations begin to decrease, more of it is produced from beta- hydroxybutyrate” (source). As a result of the continued conversion of acetoacetate to beta- hydroxybutyrate, the serum and urine volume of acetoacetate (the only ketone detected by ketostix) is significantly reduced. Your takeaway point is this: If you stay on your keto diet for long enough, the primary ketone circulating in your serum (and consequentially, present in your urine) is not detected by your ketostix. ![]() ![]() ![]() ![]() ![]() ![]() Why You Need To Stop Worrying About The Color Of Your Ketostix Posted on Sunday, September 9th, 2012 at 7:42 am. Fox 5 NY, New York News, Breaking News, weather, sports, traffic, entertainment. You’ll recall from last week’s post I did a self-experiment to see if I could learn something about the interplay of exercise and ketosis, at least in myself. Now in remission, Jake faces new challenges - rebuilding his body and his life. MSN Health and Fitness has fitness, nutrition and medical information for men and women that will help you get active, eat right and improve your overall wellbeing. Reduction in kidney excretion of ketones. Besides the change in relative volumes of serum ketone types, as you become keto- adapted your kidneys naturally down- regulate the volume of ketones they excrete into your urine. Ultimately suggest that it is the result of kidney adaptation to sustained carbohydrate restriction (1. Even though we lack the sophisticated understanding of why kidneys downregulate ketone secretion into urine, we know that it happens and we know that the volume of acetoacetate in your serum drops as you become more fully ketoadapted. So quite literally, after a few weeks into your low carb ketogenic diet, you’ll likely start seeing a change in your ketone levels as measured by ketostix. If you go this route, you’ll also have to buy their very expensive and proprietary Precision Xtra Ketone Test Strips to use with it. And when I say expensive, I’m serious. You’re looking at ~$5. So you can spend the money and keep an eye on your ketone levels by measuring serum levels, or you could just not spend money on ketostix and expensive testing gizmos. The interplay of exercise and ketosis – Part II - You’ll recall from last week’s post I did a self- experiment to see if I could learn something about the interplay of exercise and ketosis, at least in myself. To understand this discussion, you’ll want to have read Part I of this post. However, before getting to this, I want to digress and briefly address two unrelated issues: Some of you (about 6. Harvard’s School of Public Health. Here is the summary of the results from my personal experiment: Let’s take a closer look at what may have been going on in each workout and see what we can learn. Swimming. This workout probably produced the most lactate of the three workouts (we don’t know for sure because I only measured immediate pre- and post- levels without measuring in- workout levels). In reality, cardiac myocytes (heart muscle cells) also “like” B- OHB more than glucose and probably also access it when it is abundant. Lactate levels by the end of the ride were effectively unchanged though. Based on “feel,” I suspect I hit peak lactate levels of 8 to 1. M on this ride during peak efforts, but I had ample time to clear it. A few observations: I consumed 6. Generation UCAN’s super starch), yet this did not appear to negatively impact my ability to generate ketones. I’ll write a lot about these later, but for now just know the CP system is good for very short bursts of energy (say 1. I do not believe either accounted for the sharp rise in blood glucose and, again, I believe hepatic glucose output in response to a strong catecholamine surge attributed to this increase. Lactate levels also rose, though probably less so than during a peak swim effort. But I have a very hard time believing 3 or 4 gm of BCAA could suppress B- OHB. When you see hoof prints in the sand, you should probably think of horses before you think of zebras. Conversely, there is some evidence that lactate promotes re- esterification of fatty acids into triglycerides within adipose cells. High levels of lactate take free fatty acids and help promote putting them back into storage form. In other words, we may be missing this effect because of my sampling error – I only sampled twice per workout, rather than multiple times throughout the workout. I think it’s safe to say I did not definitively answer any questions, which is not surprising given the number of confounding factors, lack of controls, and sample size of one. Take the following example: Someone my size can probably store about 1. This represents about 1. I can store at any one time. Before I was ketotic, you’ll recall from my post on the change in my physical performance, my RQ at 6. VO2 (about 2,5. 00 m. L of O2 per min consumption) was nearly 1. I can hold for hours from a cardiovascular fitness standpoint) I required 9. Now I only store 5. In ketosis, my RQ at 6. VO2 is 0. 7. 7 (at last check), telling me I am getting only 2. Just consume more glucose as you ride! It’s not that simple, but you’ll have to wait until my upcoming post, “What does exercise have to do with being in the ICU” to find out. Going back to the black sheep example I open Part I of this post with, we know that at least one person in nutritional ketosis seems to make enough liver and muscle glycogen to support even the most demanding of his energetic needs.
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